ANTHROPOMETRIC MEASUREMENTS AND ORAL HEALTH: WHICH IS THE COMMON LINK ? Furthermore, although the correlation between fructose consumption and blood pressure values was not statistically significant, the definitely hypertensive children (blood pressure ≥95th percentile) were among the main consumers of sweetened beverages. Results of our paediatric pilot study overall showed that grams of sugar per week and monthly and daily frequency of sugary drink consumption, reflect the excess body weight and metabolic syndrome clinical parameters, with the maximum intake per serving (>3 glasses per time) in 50% of the obese group. Additionally it has been reported that fructose is associated with metabolic syndrome components including increased systolic blood pressure and insulin resistance. This depends in particular on the lack of satiety after sweetened beverages which don’t influence food intake.
#Anna gunn weight gain 2017 series
Pediatric RCTs interventions comprising or including recommendations to reduce sugar-sweetened foods and beverages, instead were in general characterized by low compliance and no change in body weight.įructose consumption is therefore a key contributor to the obesity epidemic, particularly in paediatric ages, as demonstrated by a series of studies that focused especially on sugar-sweetened beverages (e.g., soft drinks, commercial fruit juices, energy drinks, iced teas).
#Anna gunn weight gain 2017 free
Meta-analyses of randomized controlled trials (RCTs) in adults suggests an association between reduction of free sugars intake and reduced body weight in line with a comparable increase in body weight associated with an increased intake of free sugars. Global consumption (A) and drawing stocks (B)of sucrose for 2016/17Īdded sugars supply food energy but no other nutrients (empty calories) andincrease risk of developing obesity, cardiovascular disease, hypertension, obesity-related cancers, and dental caries. Here we will focus mainly on the pandemic sucrose consumption and obesity. Īlso gut microbiota dysbiosis and altered intestinal permeability have recently been proposed to play a relevant role, and be a possible target for treatments of those cases unresponsive to diet and reduction of sedentary life. Bisphenol A ), are emerging key factors most likely involved in the onset and treatment-resistance of obesity and obesity-related complications.Ī pilot study focusing on these 3 factors in the Salerno’s area confirmed that they are additional suitable targets for future preventive and/or therapeutic approaches in paediatric obesity. exposition to endocrine disruptors (e.g. In recent years mounting evidence has suggested that The unavailability of other ancillary pathomechanism-driven effective treatment strategies or better-tolerated treatments remains one of the main problems. Also the standard lifestyle changes-based treatment is flawed by high failure and/or relapse rates due to poor compliance at all ages. Obesity prevention in the paediatric age has been the object of several efforts at all levels but it has hardly obtained efficacious results.
Since rates of overweight and obese adults and children in recent decades have dramatically increased worldwide, the World Health Organization (WHO) has classified this trend as a real obesity “epidemic” and the American Medical Association changed the term obesity from a condition into a disease in 2013.
Obesity is a chronic disease characterized by a multifactorial aetiology with a complex interplay between genetic susceptibility and environmental factors.
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